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Gene therapy research for MEF2C haploinsufficiency syndrome

Gene therapy research for MEF2C haploinsufficiency syndrome

Status
Active, not recruiting
Phases
Unknown
Study type
Observational
Source
ChiCTR
Registry ID
ChiCTR2600127368
Enrollment
Unknown
Registered
2026-06-30
Start date
2026-07-01
Completion date
Unknown
Last updated
2026-07-13

For informational purposes only — not medical advice. Sourced from public registries and may not reflect the latest updates. Terms

Conditions

MEF2C (Myocyte Enhancer Factor 2C) haploinsufficiency syndrome is a group of clinical syndromes caused by mutations in the MEF2C gene or microdeletions on chromosome 5q14.3, leading to abnormalities in the MEF2C gene. Its pathogenesis is that after gene mutations or deletions, only 50% of the normal level of protein is produced, which is insufficient to maintain normal physiological functions of c

Interventions

control group:None
Patients with mef2c haploinsufficiency (Haploinsufficiency of MEF2C gene (pathogenic variation/deletion of MEF2C gene)):None

Sponsors

Children's Hospital,Zhejiang University School of Medicine
Lead Sponsor

Eligibility

Sex/Gender
All
Age
1 Years to No maximum

Inclusion criteria

Inclusion criteria: 1.Patients with MEF2C gene mutations;

Exclusion criteria

Exclusion criteria: 1.Combined with other known syndromic neurodevelopmental disorders (such as Rett, Angelman, Fragile X, etc.) or chromosomal abnormalities (>5 Mb deletion/duplication); 2.Received gene therapy, cell therapy, or immunomodulatory therapy; 3.Unable to obtain informed consent from legal guardian;

Design outcomes

Primary

MeasureTime frame
MEF2C protein and mRNA expression level;

Secondary

MeasureTime frame
Neuronal differentiation efficiency;Neuronal electrophysiological activity;

Countries

China

Contacts

Public ContactYue Zhang

Children's Hospital,Zhejiang University School of Medicine

yue.zhang@zju.edu.cn+86 571 86670083

Outcome results

None listed

Source: ChiCTR (via WHO ICTRP) · Data processed: Jul 23, 2026