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Study on the Mechanism of Increased Pulmonary Lactate Promoting LPCAT2 Lactylation Modification Aggravating Acute Lung Injury in Sepsis

Study on the Mechanism of Increased Pulmonary Lactate Promoting LPCAT2 Lactylation Modification Aggravating Acute Lung Injury in Sepsis

Status
Active, not recruiting
Phases
Unknown
Study type
Observational
Source
ChiCTR
Registry ID
ChiCTR2500101899
Enrollment
Unknown
Registered
2025-04-30
Start date
2025-03-03
Completion date
Unknown
Last updated
2025-05-05

For informational purposes only — not medical advice. Sourced from public registries and may not reflect the latest updates. Terms

Conditions

Acute lung injury in sepsis

Interventions

Normal group of patients:None
Sepsis acute lung injury group:None

Sponsors

Shenzhen Hospital of Southern Medical University
Lead Sponsor

Eligibility

Sex/Gender
All
Age
18 Years to No maximum

Inclusion criteria

Inclusion criteria: The diagnosis of sepsis and septic shock follows the Third international consensus on the sepsis defense law The criteria outlined in (sepsis-3). If the patient is over 18 years old and after admission Within 24 hours, they are eligible to be included in the study.

Exclusion criteria

Exclusion criteria: Exclusion criteria included pregnancy, immunosuppression, leukopenia, and hematologic malignancy tumor or initiation of palliative care.

Design outcomes

Primary

MeasureTime frame
Lactate;inflammatory factor ;Oxygen coefficients;—

Countries

China

Contacts

Public ContactYoutan Liu

Shenzhen Hospital of Southern Medical University

youtanliuhao@163.com+86 139 0244 8143

Outcome results

None listed

Source: ChiCTR (via WHO ICTRP) · Data processed: Feb 4, 2026