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Study on the mechanism by which fatty acid transporter CD36 improves myocardial insulin resistance induced by pressure overload

Study on the mechanism by which fatty acid transporter CD36 improves myocardial insulin resistance induced by pressure overload

Status
Recruiting
Phases
Unknown
Study type
Observational
Source
ChiCTR
Registry ID
ChiCTR2500099708
Enrollment
Unknown
Registered
2025-03-27
Start date
2025-01-08
Completion date
Unknown
Last updated
2025-03-31

For informational purposes only — not medical advice. Sourced from public registries and may not reflect the latest updates. Terms

Conditions

Myocardial insulin resistance induced by pressure overload

Interventions

Dilated cardiomyopathy patient group:None

Sponsors

Tongji Hospital, Tongji Medical College ,Huazhong University of Science and Technology
Lead Sponsor

Eligibility

Sex/Gender
All

Inclusion criteria

Inclusion criteria: Dilated cardiomyopathy patients undergoing heart transplantation/myocardial biopsy.

Exclusion criteria

Exclusion criteria: None

Design outcomes

Primary

MeasureTime frame
Content and location of InsR;Content and location of CD36;Content and location of HSP90;—

Countries

China

Contacts

Public ContactFan Jingjing, Hongyang Shu

Tongji Hospital, Tongji Medical College ,Huazhong University of Science and Technology

shy@tjh.tjmu.edu.cn+86 27 8366 2483

Outcome results

None listed

Source: ChiCTR (via WHO ICTRP) · Data processed: Feb 4, 2026