None listed
Conditions
Brief summary
The goal of this study was to investigate the association between second hand smoke exposure and sleep bruxism in children. Nicotine is known to induce acetylcholine and glutamate synaptic transmission and enhance dopamine release. In smokers, nicotine accumulates in the body during the time spent awake, decreasing gradually during sleep. Higher levels of smoking, leading to increased levels of nicotine and dopamine release, could thus be related to increased oromotor activity.
Interventions
Sleep bruxism was analysed in children of group 1 after six-months of not being exposed to second-hand smoke. The smoking members of the families in group 1 were asked not to smoke in the presence of the child for a period of six months. Parents were told prior to study enrolment that they would not be required to quit smoking as part of study participation; rather they would be asked to eliminate children second hand smoke exposure. The study does not include parents who do not smoke. Children with bruxism exposed to second-hand smoke and then not exposed for 6 months ( same children, same families) were compared with a control group of children with bruxism exposed to second-hand smoke for 6 months. Some families selected in group 1 refused to participate in the trial because, despite being informed about the risks of SHS, they reported that they would not have been able to reduce SHS exposure for their children.
Sponsors
Study design
Eligibility
Inclusion criteria
Children from elementary schools.
Exclusion criteria
Cleft lip and palate; disability, respiratory disorders; orthodontic and/or orthopaedic treatment.